Marshall Edwards, a subsidiary of the Australian pharmaceutical company, Novogen Limited, was formed with the specific intent of increasing the focus on an important new oncological discovery known as multiple signal transduction regulators (MSTRs).

Novogen scientists discovered these regulators, and brought Marshall Edwards onboard to develop the products commercially. The main MSTR, called Phenoxodiol, is currently undergoing Phase III clinical testing in the US, Europe, and Australia. The drug is being developed for the treatment of both early and late-stage prostate and cervical cancer, as well as ovarian cancers that have proven to be resistant to chemotherapy.

Phenoxodiol promotes the death of cancer cells by inhibiting harmful proteins which prevent apoptosis (the natural process of self-destruction in certain cells). The drug can be used in conjunction with other, more traditional forms of treatment.

A feeling of cautious excitement comes with every possibility of reducing or eradicating cancerous cells. What sets Marshall

TapImmune, Inc. (OTCBB: TPIM) announced that their TAP incorporated vaccine for the treatment of cancer was recently reviewed by a third party research article in the Journal of Investigative Dermatology, which confirmed that the company’s vaccine should be considered as a therapy for cancer.

The article, entitled, “Restoration of the Expression of Transporters Associate with Antigen Processing in Human Malignant Melanoma Increases Tumor-Specific Immunity,” was published in advance online on April 3, 2008. TapImmune is a biotechnology company that specializes in the development of immunotherapeutics for cancer and vaccines for infectious diseases, and the company’s stock rose 37.10% in Monday’s trading.

The company’s promising TAP vaccine performs a key step in moving characteristic markers called antigens to the surfaces of cells. TAP, which stands for Transporters with Antigen Processing, are proteins responsible for supplying tumor-associated antigens (markers) and viral antigens to the surface of infected cells. Without